Hedblom Syndrome

Imagine that you are a doctor and you discover a patient with a rare form of disease that occurs in less than one percent of patients. The young guy suddenly became numb in his upper limbs for no apparent reason. You suspect headball syndrome, but don't know how to confirm or treat it. This situation may seem unpleasant and inconvenient, but this is where I would like to stop and talk about what I know about this disease. In addition, I will try to suggest possible treatment options and diagnostic options for this condition.

Headball syndrome is a rare, virtually unstudied disease that is characterized by loss of sensation in the upper extremities and impaired motor skills of the fingers. At the same time, patients often complain of frequent headaches and muscle weakness. In rare cases, loss of vision, impaired coordination of movement, and problems in the functioning of the cardiovascular system are possible. At the beginning of the diagnosis, a detailed conversation with the patient and a neurological examination are necessary to confirm the diagnosis of Hedblem syndrome. It is important to remember that the disease is under study, and additional diagnostics may be required to obtain more accurate information about the disease process. Treatment of this disease begins with identifying hidden problems that can provoke the manifestation of Headlom symptoms.

Hedblom syndrome, headache-wandering disease, “ball disease”, chronic head pain syndrome (headache moving pattern syndrome) is a symptom complex that includes repeated paroxysmal, often relieved by analgesics (up to frequent, “often” - every week or more often) episodic compressive headaches lasting from several minutes to an hour, which have the ability to be independently transferred from one area of ​​the head to another, mainly from one slope (on one side) of the skull to the other and around the head, usually without clear boundaries between the sensation of the head from the outside and from the inside; the term “head-wandering” refers to the fact that pain paroxysms are often described by patients as if “headache is wandering around the head” (hence the term “headache” itself is sometimes transferred to areas of the body that are not directly related to the head). Typically, when the patient lies motionless on his back or on his side during an attack, the pain moves down and to the left only from the beginning of the paroxysm, passing through a spontaneous reverse movement of pain up and to the right as the pain paroxysm begins to fade. The most common explanation for the syndrome is that vagal compression occurs because this globular brain tissue is functioning very close to its source, and these pressure pulsations block the flow of cerebrospinal fluid in the skull. Brain tissue consists of clusters of various bodies (neurons and glial cells). It is believed that the pulsation of dural vessels and pulsating pressure waves are generated by the operating networks of these clusters. Internal (basilar) pulsation from the vessels of the pia plexus, rhythmically pressing on the body of the cerebellum, in combination with the pulse caused by contractions of the internal membrane, which are located in the portal system, helps to create a hyperemic and enlarged gelatinous smear of the medulla, which creates continuous pressure impulses, initiating wandering pain. As a result, if hyperemia occurs in two different centers located in the same hemisphere of the brain, these two separate