Oculomotor Nerve Syndrome

Oculomotor nerve syndrome is diagnosed annually in a significant portion of the adult population. This is a chronic disease that requires regular medical monitoring. The most noticeable sign is eye problems such as eyelid ptosis and strabismus. This nerve syndrome is caused by various factors, such as long-term smoking, frequent stress and thyroid disease. In this material we will talk about the dangers of oculomotor nerve syndrome and what symptoms may indicate the presence of this disease in a person.

What is Oculomotor nerve syndrome? Oculomotor nerve syndrome, also known as Bettianacker neurosis, is a complex disorder caused by disruption of the normal functioning of the extraocular muscles. It manifests itself as a loss of normal movement or stability of the eye muscles, which

Oculomotor nerve syndrome is a set of symptoms associated with dysfunction of the oculomotor nervous system. The oculomotor nerve ensures the movement of the eyeballs up, down, left and right, as well as blinking and coordination of these movements. This area is nervous

Oculomotor (III) nerve syndrome is a rare disease characterized by various combinations of paralysis of the external, less often internal rectus and oblique muscles of the eyeball with damage to all or several branches of the oculomotor nerve. There are 3 forms of oculomotor paresis: peripheral, central and mixed. Symptoms of peripheral oculomotor paralysis are always more pronounced with damage to the VI nerve, and the central - to the III nerve. It is characterized by facial atrophy, swelling of the conjunctiva, and the eyes gradually lose mobility. Visual acuity is sharply reduced.

The ophthalmologist's or neurologist's problem may appear in the form of oculomotor nerve syndrome (OMN) and is characterized by refractive errors of the eye and other structures. The syndrome accompanies a lot of eye, vascular and other diseases of the nervous system. The causes of the problem are a large number of nerves that control the movements of the eyeball and the functioning of the iris sphincter. So it turns out that as eye pathologies develop, they are eliminated incorrectly, which is the basis of the SGBV clinic. A number of instrumental and laboratory methods will help in diagnosing SGBV. But there are “classic” symptoms for diagnosis. These are common signs for neuro-ophthalmology: redness of the facial skin and dilation of the blood vessels in the eyes, increased reaction of the pupils to bright light and tactile irritation of the eye shell, increased tear flow, etc. Based on this, the doctor will determine what specific oculomotor pathology is provoked by SCH, and what treatment will be most effective. Also, the symptoms of oculomotor nerve syndrome are similar to the manifestations of damage to the abducens nerve, drooping eyelid, strabismus, as well as paralysis of the muscle on one side (with paralysis of that half of the motor apparatus of the eye, the lower eyelid does not cover it) or compression and narrowing of the pupil (it develops spotism and shine ). If a third of the nerve is damaged, double vision develops when looking down. Partial SGB causes opacities in the pupil, iridodynia, visual disturbances and other manifestations. As for the reasons for the development of oculomotor nerve syndrome, they are different for everyone. Among them: syphilis, drug complications, tuberculosis, brain injuries, abscess, cancer, meningitis, typhus, rabies, lead poisoning, intoxication, brain infections, etc.

The ocular muscles are innervated by 3 pairs of cranial nerves, the 4th by nerve fibers to the fibers of the auxiliary apparatus of the eye. Symptoms of gaze paresis can be divided into upper and lower. Upper symptoms include upward or downward deviation of the eyeballs.

Inferior symptoms include inward or outward deviation of the eyeballs or a vertical alignment reaction. This criterion may be useful in persons with diplopia of unknown etiology. Oculomotor nerve syndrome manifests as: staring, avoiding gaze, facial palsy, eye weakness