Believers Autopsychosis

Wernicke's autopsychosis is a disorder of the temporal cortex of the brain, which is associated with memory, speech comprehension and language skills. People suffering from this disorder may have confusion about words, difficulty understanding other people's speech, and trouble understanding written language and contextual information.

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*Wernicke/Vennikov autopsychosis is a symptom complex caused by bilateral damage to Wernicke's area in the left temporal lobe and combined with motor and sensory aphasia, as well as amnestic disorders. The discrepancy between preserved expressive, objective speech, intelligence, non-pessimistic mood and lack of criticism, characteristic of the syndrome, is the main diagnostic evidence; it makes it possible to distinguish V. a. from mental retardation, schizophrenia and depression.*

***Etiology***. The main source of the disease is acute alcohol poisoning (in particular methyl alcohol) in the absence or insufficiency of the gag reflex and anamnestic indications of a toxic-allergic reaction to alcohol poison (methanol) from the cardiovascular system and gastrointestinal tract. Possible infectious-toxic (botulism, neuroborreliosis, tetanus), as well as hypoxic (in cardiovascular accidents) origin. Based on a comprehensive analysis of epidemiological and follow-up data, the authors proposed the following scheme for the clinical course of V.a: 3% - fulminant form, 2.5-4% - short (with a quick outcome - from the end of the first to the beginning of the second week), 24- 30% - protracted, widespread, deep and plastic (5-14 days), 40-50% - chronic, phase-recurrent with rare remissions, benign (at least half a month) and less plastic than the previous one; the remaining 30-35% are erased and special (tumor and diencephalic) forms. It should be noted that patients abroad, due to lacunar changes in speech and speech defects, very often underestimate the severity of their illness. Their motivation for seeing a doctor is, for example, the thought of disease progression (which influences the decision to amputate) or the desire to confirm the presence of a mental disorder (after talking with a doctor, they have a fear of losing their job). Many drug addicts, in whom alcoholic psychosis occurs with a predominance of striopal symptoms and is usually combined with delirium, describe their motives for admission to the hospital in a similar way. In oncology departments and in therapeutic hospitals, in connection with the phenomena of withdrawal syndrome, the same thing is observed in heroin addicts (the desire to undergo a course of abstinence treatment in order to start work or study). For psychiatric surgeons, one of the main diagnostic signs of V. a is the absence of a reaction to propofol or fentanyl against the background of severe alcohol intoxication (a frequent moment for suggesting anesthesia and refusing surgery, but the need to continue “treatment for alcoholism”). In heavy alcoholics without AAD, anesthesia-induced sleep can only be induced by barbiturates.

In persons with severe alcohol poisoning, there is a deficiency in the detoxification function of the liver, a tendency to hemoconcentration, and severe leukocytosis. Features of the cellular composition of peripheral blood

Wernicke's auto-psychosis is a psychotic condition that can occur in a person with damage to the posterior part of the inferior temporal gyrus in the left hemisphere of the brain. This disorder may manifest as hallucinations, delusions, and perceptual disturbances. Wernicke's disease can develop in a variety of situations, including head trauma, intoxication, thyroid disease, and other conditions. Here is an article about this phenomenon:

Wernicke-Curie autopsychos is a mental symptom associated with a state of stress and excessive excitement of the psyche, especially characteristic of temporal brain damage (for example, hysterical nature). V.K. usually appears in parallel with twilight stupefaction and has the character of irritable weakness, confusion and absurd fear, alternating with an affect of anger and aggressiveness. Ernst Hopffer syndrome was first described by E. Hopffer in 1893. In 1918, Wilhelm Griesinger suggested that the cause was organic damage to the brain, and H. Fuchs saw in this condition disturbances in the transmission of nerve impulses through the hippocampus. The theory of E. Garcia and T. Bernstein is the hypothesis of functional disorders of the autonomic nervous system with intense pathological excitation of the centers of the limbic system. The temporal focus of consciousness disorders is “laterality of dominance” according to A. R. Luria. There are no somatic disorders of muscle tone or signs of hypoglycemia. A characteristic feature is a change in the state of sleep: after 2–3 days of absence of consciousness, hallucinatory experiences occur. The main pattern of development is a staged sequence: severe confused pseudodementia - then an episode of dysphoria and a psychopathic symptom complex - subsequently severe verbal hallucinatory psychoses appear with a protracted course and intermittent preservation of consciousness in the form of a nightmare and illusions - consciousness acquires fragmented dynamics, visual illusions can occur even during clear consciousness. The condition is characterized by affective tension and episodes of maliciously aggressive behavior, and catatonic stupor reflexion is possible. Distinctive features are changes in affect and mood, the presence of verbal hallucinations, and the predominance of the affect of anger. Complete or incomplete amnesia during the period of stupefaction is possible. Usually the first seizure is paroxysmal in nature - there is a cry, excitement in the facial muscles, a feeling of horror arises, a transition to oneiroid appears with dream-like and hallucinatory experiences (shadows, soft objects, feelings of intoxication and weightlessness), possibly convulsive enticement. Affective disorders are noted, general paroxysmal fluctuations in mood and affective balance are noted - amnestic-dysmnestic features. Epileptic paroxysms usually begin with an attack of clonic-tonic seizures with