Broca's Ichthyosiform Dermatosis

Broca's ichthyosimorphic dermatosis - description of the disease The skin disease appears due to an error in the functioning of the immune system and is characterized by thickening of the epidermis. The pathology is non-infectious, that is, damage to the body by a viral, bacterial or fungal nature is excluded. The disease is considered hereditary and usually affects men. The cause and mechanism of development of the Broca ichthyosymorphoform are still not completely clear. Scientists suggest that the reasons may be:

- stress and overstrain of the whole body; - deficiency of certain vitamins (A, E, D); - periodic metabolic disorders; - constant exposure to the street (solar insolation); - chronic diseases of internal organs. *The course of the disease is extremely complex and depends on many factors. It is very important to exclude all possible allergens and minimize the influence of external irritants. If the course of the disease coincides with a flu epidemic, you should immediately consult a doctor to avoid complications.*

Brock rodimfokaich tis layal tnemiy photochny (18.56 - 19.28), German dermatology

Brocq, L.A.J., 1875. Lichen bullosus erythrodermic (psoriasis herpetiformis). Virchows Archiv 32, 464-527. Brock; L.A.J. Lichenosunlariodermal (Herpetine perctiformia Psora)

Brock's phenomenon:

Brockmann syndrome. Brocke was a German dermatologist who described skin changes called psoriasis herpetinum in 1862. This term has been proposed to describe a lesion of the skin or other parts of the body that involves red patches covered with white scales and other changes. These lesions usually affect the skin of the arms, legs, neck, face and buttocks. He predicted that it might be due to a virus and suggested that the desquamation was due to an increase in the number of keratinocyte cells. However, since the disease lasts for a long time, the exact mechanism is unknown.

Identification of Brockmann syndrome

By the early 20th century, it was believed that herpetin psoriatic disease, described by Brocca, was the only known form of psoriatic disease (also called herpetin psoriatic dermatitis). Today it is known that there are many forms of psoriatic disease, each with its own clinical features and etiology.

In recent years, new forms of the psoriatic process have been discovered. Among these forms, "herpetine psoriatic condition" has also been discussed. During the study, in the non-exudative stage of the disease, Brokke noticed the deposition of a substance rich in carbohydrates. This substance was later named type VI glycoprotein when its antibodies were discovered in patients with herpes types I and II. Professor Erhard Bunge, who teaches neuropathology and dermatology, was very close to the discussion of new forms of the disease. He developed “inflammation” as a psychogenic endocrine irritation, contributing to the reappearance of secondary sexual hair, as well as the processes of varicose veins and platelet-derived cutaneous albuminuria, especially during exacerbation of the disease.

Today, Brockmann syndrome is considered a variant of white scaly plaque. In clinical practice, it is no longer known under the name "Simple psoriatic process", since its histological pathogenesis is known, the clinical symptom has a more solid basis.

The clinical characteristics of Brockmann syndrome today can be characterized as follows: - During acute paroccium, a symptomatic, bluish-red