Fetopathy Toxoplasmosis

Toxoplasmic fetopathy (phosphoplasmomia). Form of congenital insufficiency: with hydrocephalus - disruption of metabolic processes and blood flow in the central nervous system, resulting in insufficiency and swelling of the brain due to paresis of the brain, hemorrhage in the brain structures or intracerebral hydrocephalic cysts of the brain. Occurs from the moment of infection to the eighth week of pregnancy; danger is especially likely in the second trimester. Parasitism by T. ophthallam production affects the brain, the nervous system of the child through the blood-brain barrier, intrauterine changes in the placenta, and infection of the fetal eye organs. Hernias of the central nervous system with open neuroinfections are provoked by the herpes virus in elderly people and with TORCH infections. The cause of genetic pathologies is infection of the expectant mother with Toxoplasma, regardless of the type of disease. The risk remains with recovery after three years. The deviations are most pronounced in infants. With congenital hydrocephalus, symptoms increase gradually from birth to six months. Primary brain damage has no clear signs and is expressed by headache in the frontal region, vomiting, lethargy, drowsiness, convulsions, pressure fluctuations, and hallucinations. In the second half of the year, the baby becomes more restless with muscle hypotonicity. At one and a half years of age, developmental delays are observed. A rash or spots appear. A distinctive sign of toxoplasmosis in infants is impaired brain activity with frequent attacks of epilepsy. Due to damage to the cervical spinal cord, sleep develops