Myosiderosis

Myosiderosis (also known as Rossolimo-Locas syndrome) is a rare autoimmune disease in which the immune system attacks muscle fibers, causing inflammation and muscle damage.

Myosiderosis is caused by the body's own antibodies, called autoantibodies. These autoantibodies can bind to various components of muscle tissue, including actin, mitochondria, and other cells. The result is inflammation and destruction of muscle tissue.

The first symptoms of myosiderosis may appear as muscle weakness and difficulty moving. Over time, the condition worsens, the muscles become weak, painful and atrophy. Patients may experience muscle cramps, abdominal pain, and fatigue.

Treatment may include medications such as glucocorticoids or antitoxins to reduce inflammation and repair muscle tissue. Surgery can also be used to remove damaged muscle fibers. However, there is no complete cure for myosiderosis, and the disease can recur.

Myosiderosis is a fairly rare disease, but it significantly affects the quality of life of patients. Therefore, the diagnosis and treatment of this disease requires integrated approaches and interdisciplinary cooperation between specialists of various profiles.

Myosideratosis (synonym: fibrous dysplasia).

Fibrous dysplasia (abbr. f/d) is a hereditary disease of connective tissue, mainly skeletal muscles and bones. It is systemic in nature (several human organ systems are affected). However, the muscular type of the disease is the most common. In turn, it can occur almost everywhere, regardless of gender and age.

Etiology. Predisposition