Tachycardia Ventricular

VENTRICULAR TACHYCARDIA Ventricular (sinus) tachycardia (Tachycardia ventriculare sinoauricularis, English) is an increase in heart rate (HR) up to 200 per minute or more. It is also called supraventricular tachycardia (tachyarrhythmia), since a violation of the conduction of impulses through the sinus node, and not from the ventricle, develops. In some cases, tachycardia is caused by an ectopic rhythm, often caused by early or complex extrasystoles.

There are tachycardias, including those provoked by physical exercise. Tachycardia is usually referred to as an increase in heart rate because only 35–45% of S–T interval prolongations are due to myocardial ischemia. A number of authors argue that tachycardia as such does not exist from the ECG point of view: it is a group of sinoauricular arrhythmias that differ from tachycardias associated with the conduction of impulses through the AV node. Based on the time of occurrence, tachycardia is also divided into supraventricular, occurring in the sinus node, and nodal, developing outside the sinus node. Among the causes of tachycardia, strong emotional stress, fever (at the time of an increase in body temperature, heart rate can increase to 40–60 beats/min for several minutes and return to normal when the body cools, i.e., with the restoration of normal temperature), cardiac intoxication glycosides and (cautiously) digitalis, hypovolemia (after the administration of intravenous fluid, the systolic volume of the veins increases several times), loss of chlorides, catecholamines, adrenal hubbub, and with the so-called. thyrotoxicosis, but usually tachycardia is caused by dysfunction of the sinus and AV nodes. Previously, ventricular extrasystole was isolated, and it was believed that this atrial and atrioventricular rhythm does not differ significantly from sinus tachycardia: changes in the electrocardiogram correspond to the selection of a curve showing different arrhythmias, although we are talking about different rhythms. The appearance of specific changes on the FCG indicates the presence of a left-sided Müller reflex (i.e., deviation of the electrical axis of the heart to the right) and serves as confirmation of a flattened wave