Lymphocytic choriomeningitis virus

Lymphocytic choriomeningitis virus (LCM) is one of the most dangerous viruses of the Arenavirus family, which causes disease in humans and animals. It belongs to the arenavirus genus and has some similarities to the hemorrhagic fever virus.

PCM can be transmitted through contact with infected animals or through blood. People infected with LCM may develop a latent infection that can last for many years. This means that a person may not show any symptoms but still carry the virus.

The causative agent of LCM causes the disease of the same name in humans, which is known as lymphocytic choriomeningitis. This disease manifests itself in the form of fever, headache, muscle pain, weakness and other symptoms. It can lead to serious complications such as meningitis and encephalitis.

Treatment of LCM includes the use of antibiotics and immunoglobulins. However, because the virus can be resistant to some antibiotics, treatment can be difficult. In addition, some people may be allergic to immunoglobulins, making their treatment even more difficult.

It is important to note that LCM is a serious disease that can lead to serious consequences. Therefore, if you suspect that you or someone you know has LCM, it is important to see a doctor for diagnosis and treatment.

Lymphomachorea. Clinical picture Acute lymphocytic choriomeningitis (latent paralytic process) (Smith-Fredenberg disease) is characterized by manifestations of an acute intracranial infection caused by the lymphocytic choriovirus and occurring among middle-aged people, in the absence of any previous viral disease or disorder of the immune system. The virus is transmitted through the blood of an infected partner who is in the late or spermatogenic period (or from mother to fetus). Patients either report no symptoms or symptoms that are mild or moderate and may last several months. These include seizures, brain spasms, loss of consciousness, etc. The picture of cerebral LCM can be mistaken for that of encephalitis or myelitis caused by encephalomyelitis or a spinal cord tumor. If the patient undergoes a cranial x-ray, it may reveal enlargement of the cerebellum or hyperopia of the internal parts of the skull, including the cerebral diaphragm (subependymal edema) or rupture of the arachnoid membrane. MRI is often particularly helpful in detecting these changes. Other signs that may appear in patients with CCLM include spasticity, blood eosinophilia, ocular syndromes, and psychiatric disorders such as hallucinations, depression, or psychosis. Some patients may develop normal cerebrospinal fluid, while others may have high fluid levels. Examples Approximately half of the thousand young people infected with primary latent lymphocytic choreameningitis develop a condition of increased sensory involvement of the carotid artery, also known as increased sensory involvement of V4 [32], [33]. Abnormalities in the fifth cortical area, usually associated with lateral inflammation or cerebral hemorrhage described in the patient's medical history, explain why normal patients begin to appear impaired after acute symptoms. The dominant and non-derived regions of the temporal lobe and even the basal ganglia are affected. Temporal lobe lesions were most prominent in normal patients [34]. In patients with latent lymphocytic choreameningolithia, after lethal events in the cerebral cortex, changes in the temple were observed. The perception of time as area 8, located in the posterior part of the striatum. Damage to the temporal cortex was more pronounced in childless patients [35]. Atrophy or pitting of the temple was most noticeable in patients with