Hyperbilirubinemia Podpepenochnaya

Subhepatic hyperbilirubinemia: Causes, symptoms and treatment

Subhepatic hyperbilirubinemia, also known as posthepatic hyperbilirubinemia, is a condition in which the level of bilirubin in the blood is elevated due to abnormalities in the hepatic system. Bilirubin is a yellow pigment produced by the breakdown of red blood cells, and its normal removal from the body is ensured by the liver. However, with subhepatic hyperbilirubinemia, liver function is impaired, which leads to the accumulation of bilirubin in the blood.

The causes of subhepatic hyperbilirubinemia may be different. One of the most common causes is an obstruction in the biliary tract, which can be caused by gallstones, tumors, strictures, or congenital abnormalities. Hyperbilirubinemia may also be associated with inflammatory liver diseases, such as cirrhosis or hepatitis, or with gallbladder pathologies.

The main symptom of subhepatic hyperbilirubinemia is jaundice, in which the skin, sclera of the eyes and mucous membranes become yellow due to high levels of bilirubin in the blood. Other symptoms may include malaise, fatigue, loss of appetite, nausea and itchy skin. If hyperbilirubinemia is not treated, it can progress and cause complications, including liver damage and impaired liver function.

The diagnosis of subhepatic hyperbilirubinemia is based on an analysis of the level of bilirubin in the blood, as well as on clinical manifestations and the results of additional studies. Your doctor may order an ultrasound of the biliary tract, computed tomography, or magnetic resonance imaging to determine the cause of hyperbilirubinemia.

Treatment of subhepatic hyperbilirubinemia depends on its cause. If the cause is gallstones, surgical removal of the stones or gallbladder may be necessary. For tumors or strictures, surgery or other treatments such as endoscopic retrograde cholangiopancreatography (ERCP) may be required. If hyperbilirubinemia is associated with inflammatory liver disease, treatment is aimed at eliminating the underlying disease and supporting the liver. This may include the use of antiviral drugs to treat hepatitis or anti-inflammatory drugs to reduce inflammation in cirrhosis.

In some cases, symptomatic treatment may be required to relieve symptoms associated with hyperbilirubinemia. For example, anti-itch medications can help relieve itchy skin, and a diet rich in antioxidants can help improve liver function.

It is important to see a doctor to diagnose and treat subhepatic hyperbilirubinemia. Insufficient intervention can lead to disease progression and serious complications. Your doctor will conduct a full examination, determine the cause of your hyperbilirubinemia, and develop an individual treatment plan tailored to your condition.

In conclusion, subhepatic hyperbilirubinemia is a condition in which there is an increased level of bilirubin in the blood due to abnormalities in the hepatic system. Jaundice is the main symptom of this condition and treatment depends on its cause. Timely diagnosis and adequate treatment will help prevent complications and maintain liver health.

Subhepatic hyperbilirubinemia or psoas-hyperbilirubinemia is a pathological condition characterized by an increased level of free bilirubin in the blood serum. For the most part, this occurs due to increased degradation of hemoglobin in the sinusoidal spaces of the liver due to its inferiority, destruction and dissolution by the indirect fraction of bilirubin mononuclear. Thus, externally, excess bilirubin binds to the macrophages of the sinusoidal liver and is distributed throughout the body through highly vascular vessels. But this is always not enough to maintain the normal spectrum of the substance. Therefore, in psoa syndrome, a deficiency of antiporphyrin bile within the liver tissue is detected. Due to this, the removal of bile pigments from the body and the normalization of the indicator are impaired. Hyperbilirubinamia of this origin is also called exobaldial hyperbilirubinamia.