Osteogenic nephropathy

Osteogenic nephropathy (nephropathia osteogena) is a chronic progressive kidney disease that develops with common bone diseases (osteomalacia, fibrous osteodystrophy, Paget's disease), in which there is an increased excretion of bone destruction products in the urine.

The disease is characterized by impaired tubular and glomerular filtration in the kidneys due to deposition of bone tissue metabolic products, primarily calcium and phosphates, in the tubular apparatus, glomeruli and interstitium.

Clinically manifested by proteinuria, the appearance of calcium salts in the urine, hyperphosphatemia, and the development of renal failure. Diagnosis is based on identifying the underlying disease of the skeletal system, biochemical and radiological changes in the kidneys. Treatment consists of correcting bone metabolism disorders. The prognosis is serious due to its progressive course.

Osteogenic nephropathy is a disease associated with impaired renal function and the osteoarticular system. This pathology occurs due to the influence of pathogenic factors on bone tissue and renal glomeruli, which leads to disruption of the blood supply to organs, the development of inflammation and a decrease in kidney function.

Osteoneuropathy (bone marrow nephropathy) is a malnutrition of bone tissue caused by insufficient calcium metabolism. The causes of the development of the disease can be both structural anomalies and tissue metabolic disorders. In children and adults, it can manifest itself in the form of fractures, various types of deformations, pain, signs of impaired blood supply and other disorders of the body.

The disease progresses slowly, so it goes undetected for a long time. However, over time it can lead