Extrasystole of Rest

Extrasystoles of rest

Extrasystole (E) is a pathological condition of the heart muscle, which is manifested by extraordinary contractions (paroxysms of extrasystoles). Paroxysms of extrasystoles are not always associated with myocardial ischemia and can be transient or long-lasting.

The origin of E is a violation of cardiac automatism, characterized by excitation of an ectopic (normally non-existent) pacemaker. Unlike paroxysmal supraventricular extrasystoles, resting extrasystoles are of sinus origin. They arise from a normally functioning sinus node. Usually, extrasystoles are preceded by activating changes in the ECG.

Paroxysm of rest extrasystors can be considered one of the earliest precursors of an angina attack. According to I.I. Bregelman, anginal attacks that began with resting extrasystoles or other forms of arrhythmias were observed in patients in all age groups, but more often at the age of 40-50 years. In more than 25% of cases, before the development of angina, there is a previous extrasystolic rhythm destabilization of the “sinus rhythm disruption” type.

At the present stage of development of medicine, cases of extrasystole (E.) are not uncommon. This is a heart rhythm disorder, which is characterized by the appearance of additional extraordinary contractile activity of the heart. Extrasystoles can be caused by various factors. One of the causes of E. is the vagus nerve, which is responsible for

Resting extrasystoles in adults

Resting extrasystole is an extrasystolic arrhythmia with the occurrence of extraordinary contractions of the heart at rest or immediately after it, with a horizontal or sitting position of the body or in the morning and rising, especially in the morning on an empty stomach, as well as at night during sleep. Caused by activation of the sympathoadrenal system [4], the most pronounced sudden dysfunction of the sinus node is detected in the morning, after waking up on an empty stomach after 5–30 minutes. It is provoked by the position of a person, either lying upside down or lying with the leg end raised, apparently also due to his position, provokes a vertical position (turning, standing up, rising from a chair); physical, emotional overload, cooling (hypothermia); sharp pain in the heart area, vegetative-vascular dystonia of the hypotonic type - hypotension. The long-term existence of E. p. increases the sensitivity of the sinus node to catecholamines, causing the heart to hyperreact to irritants. Attacks of acute decompensation E.p. are often associated with a violation of the nervous regulation of the tone of adrenergic impulses, and an increase in heart rate activity is achieved due to the accelerated release of adrenergic receptors from the influence