Pekhrants-Babinsky

Pekhkranz-Babin syndrome (PD syndrome) is a clinical sign of pathology, often found in patients with cerebral tuberculosis. Until the end of the 70s, PD syndrome with “destruction” of brain tissue was considered as an inflammatory process, which was based on intoxication of the body due to various infections and neoplasms, i.e., the determining factor was considered to be a toxic or infectious, or age-related factor. In the early 80s. Fundamentally new ideas about the occurrence of this syndrome have emerged, and the concept of it as an autoimmune process due to sepsis, accompanied by a disorder of hemostasis, has been formulated. Babina P.I. (1982, 1985) first identified two forms of AD syndrome: enzymatic-typological and vascular. The mechanism of its development is associated with the release of vasoactive substances from hemorrhages, traumatic foci, and inflammatory plaques in post-traumatic hydrocephalus. Belova L.A. supplemented Babina’s data, proving the secondary nature of tissue breakdown in all forms of PD syndrome. The terms “built-in”, “secondary”