Kola encephalitis

Kola encephalitis: a rare infectious disease

Kola encephalitis is a rare disease that is characterized by short-term fever, followed by the development of lesions of the extrapyramidal system and (or) hypothalamic syndrome. One of the most characteristic symptoms is severe oculomotor disturbances. This disease occurs on the Kola Peninsula.

Kola encephalitis is a little-studied disease, and its causes are not fully known. However, it is known that this is an infectious disease that is caused by an unknown pathogen. It is most likely transmitted through the bites of insects such as mosquitoes.

Symptoms of Kola Encephalitis may include: headaches, fever, nausea, vomiting, seizures, incoordination and irritability. In some cases, visual and hearing impairments and speech impairments may also occur.

Kola encephalitis is a rare disease and its diagnosis can be difficult. Various laboratory tests may be required to confirm the diagnosis, such as blood tests, urine tests, X-rays, and magnetic resonance imaging.

Treatment of Kola Encephalitis consists of symptomatic therapy, which is aimed at improving the patient's condition. In some cases, the patient may need to be admitted to a hospital for observation and treatment.

In conclusion, Kola Encephalitis is a rare infectious disease that occurs in the Kola Peninsula. It is characterized by short-term fever, damage to the extrapyramidal system and (or) hypothalamic syndrome, as well as severe oculomotor disturbances. To diagnose and treat this disease, you must consult a doctor.

**Kola encephalitis** is a poorly studied disease characterized by fever, changes in extrapyramidal systems and oculomotor disorders. In Russia, Kola encephalitis occurs in the Republic of Karelia.

Symptoms of this disease were first recorded in 1922 in Northern Finland. Since then, the disease has emerged in various regions of the world. It was initially diagnosed as a variant of epidemic spinal encephalitis. However, it later turned out that encephalitis along the spinal cord causes various complications, and not just central paralysis. The main diagnostic criterion for encephalitis is diagnosis by clinical picture and spinal cord puncture. The first two days after the onset of the disease are characterized by high body temperature, vomiting, constipation and headache. During the day, body temperature drops to normal, fatigue and tinnitus appear. On the second or third day of the development of the disease, muscle rigidity appears, except for the muscles of the tongue, muscle tone around the mouth, and lack of sensitivity in the arms and legs. This occurs as a result of damage to higher nerve centers. Patients feel the aura in the form of rotation and unreality of images. The duration of the disease varies between 30, 50 or more days.

**Diagnosis of encephalitis** is carried out by methods of detecting high titers of microbial antigen in blood serum at a dilution of 1:32. It is also informative to carry out a complement fixation reaction to an antigen. To study the cerebrospinal fluid, pleocytosis (the number of lymphocytes above 250 cells in 1 mm3), eosinophilia (above 15%), increased protein content (2 or more g/l) and the total amount of protein (0.6 or more g per 1 l) are determined. . After a course of treatment, antibodies in response to this pathogen are not detected in 99% of patients.

The main cause of this disease remains the assumption of infection with the tick-borne encephalitis virus. Infection with the virus occurs more often through a tick bite, less often through aerogenic transmission of infection. High immunotolerance to viral infection is observed. Previously, encephalitis accompanied by paralysis and hypothalamus was considered an independent disease - Gettington syndrome. Now they consider encephalitis with encephalopathic syndrome and encephalitis as the etiology of the disease.

The effectiveness of treatment measures and its outcome largely depends