Supranuclear bulbar palsy

As a result of the damage mechanisms described above, complete loss or a significant decrease in motor activity of the diaphragm and expansion of the stomach may occur. If the center that regulates breathing is affected, then paralysis occurs (bulbar, laryngeal paralysis, PSPG, paralytic pseudocollapse (pseudoretraction of the stomach), accompanied by aspiration,

Paralytic syndrome of supranucular lesion of the posterior column of the spinal cord causes a combination of insufficiency of respiratory and bulbar functions. While bulbar symptoms are most noticeable on the face, respiratory disturbances become mainly pronounced months after the illness. Damage to the posterior column leads to flaccid paralysis, first of a pseudobulbar and then of a bulbar nature. The intranodal level of the lesion is associated with bulbomotor lateralization, while the extranodal location involves the body of nuclei mainly in the lower parts of the medulla oblongata and is associated with motor asymmetry that occurs with an isolated lesion of the left brainstem. Clinical signs of bulbar paralytic syndrome: central type of breathing, congestive-hyperemic face, neck and upper chest, edema of the lungs and bronchi. There are isolated episodes of apnea, flaccid and areflex bulbs without atrophy, dried out and injected with sclerosis and hyperemia vessels in the tongue. Gastrointestinal complaints in idiopathic bulbar syndrome occur only with the addition of vomiting - to the point of disgust for everything when eating any food. The disease worsens and lasts 6