Hematomyelia

Hematomyelia is bleeding in the spinal cord that can be caused by various causes. This condition can be very dangerous and lead to serious complications. In this article we will look at the main causes of hematomyelia, its symptoms and treatment.

Causes of hematomyelia

Hematomyelia can be caused by a variety of causes, including injury, infection, tumors, vascular disease, allergies, and certain medications. The most common cause of hematomyelia is trauma. The most common sites of hemorrhage are the lumbar spine and pelvis. Injuries can occur from falls, impacts, or excessive strain. Sometimes hematomyelia can be caused

Hematomyelia.

Hematomyelia is a common form of spinal compression hematoma. Occurs in 2–5% of patients who have undergone lumbar puncture in neurosurgical practice. Isolated spinal origin of hematomas (usually “pure” hematomyelia) is less common, with spinal localization accounting for about 80% of all observed spinal hemorrhages. In children, the occurrence of spinal hematoma is most often associated with semi-moral causes; in adults, extravasation from superficial veins predominates. The tortuosity of blood vessels and their branching can contribute to repeated or multiple hemorrhages. *For diagnostic purposes, this term usually means massive hematomyelopathy, accompanied by acute spinal dynamics (myeloparalysis and/or sensory disorders).* Identification of this type of hemorrhage allows, on the one hand, to distinguish it from small bruises localized at different levels of the spine, differentiate with spinal hernias and tumors, and on the other hand, clarify the diagnosis. The first visits usually occur during pregnancy (patients either apply directly during an abortion, or apply in connection with the onset of labor) or immediately after childbirth. During this period, a significant age (third decade and older) of the patients was noted, and half of them were divorced women. 40% for the first and second pregnancy, 30% for the third and subsequent pregnancies. Rarely did patients pay attention to episodes of transient paresthesia during ARVI. The majority noted pain in the lumbar region with irradiation (like referred pain) to the perineum or plexus of the lower extremities. There were no signs of meningitis. None of the patients had a history of falls or injuries. Also, taking into account the medical history, symptoms developed in the morning or afternoon. In the vast majority of patients, symptoms of lower flaccid paralysis were already observed within 1–2 hours after the onset of complaints. As the symptoms developed, their severity increased, pelvic organ dysfunction and lower atonic paralysis or total paraplegia were added. Neurological symptoms correlated with the severity of signs of plegic claudication. Neurological disorders (except for the upper extremities) usually progressed over 3 hours to 24 hours until maximum development. The depth of descending disorders ranged from complete isolated damage to the corticospinal tracts and brain connections to peripheral motor neurons, in which paraparesis was insignificant, to the stage when deep sensitivity was completely impaired. With massive hematomyelia, reduced pain zones can be observed. was carried out between the unrefined separation of hemolysis products from the plasma through blood cells and their subcapsular deposition, which is often observed for short periods but progresses. Short-term reduction of pain areas is usually usually