Bunty Hepatolienal Syndrome

Banti hepatolienal (Banti syndrome) is a rare disease characterized by dysfunction of the liver and spleen, as well as other systemic manifestations. This disease was first described by Italian pathologist Gustavo Banti in 1884.

Banti hepatolienal syndrome manifests itself in the form of an enlarged spleen and liver, as well as other symptoms such as weakness, fatigue, decreased appetite, nausea, vomiting, abdominal pain, jaundice, enlarged lymph nodes, increased body temperature and others.

The reasons for the development of banty hepatolienal syndrome can be various diseases, such as infectious diseases, autoimmune diseases, cancer, etc. Treatment of banty hepatolienal syndrome depends on the cause of its occurrence and may include drug therapy, surgery, immunotherapy, etc.

In general, banty hepatolienal syndrome is a serious disease that requires timely diagnosis and treatment.



The diagnosis of “Bunty hepatoolenal” has three forms - portal hypertension and liver failure, as well as nodular enlargements and heart failure.

Initially, this deviation was diagnosed with obstructive changes in the hepatic arteries. However, it later turned out that it was caused by changes in the left heart. Endovascular examinations of patients suffering from the anomaly revealed the presence of abnormal arteries, one of which runs next to the thyroid gland and doubles in size.

The main symptom of this deviation is severe hepatomegaly (liver atrophy often occurs) caused by portal hypertension. As a result, portohepatic veins are formed, which are often dilated. This entails an increase in nodes with spasms and an increase in the thickness of the membrane that surrounds them. Often it can be so thick that it blocks blood flow to the vessels inside. At the same time, blood exchange between the portal artery and the gastrointestinal tract and collateral channels are reduced, which further aggravates hepatomegaly.

Portal hypertension can be accompanied by various manifestations, including:

• burning in the hypochondrium;

• moderate dyspeptic disorders;

• diffuse pain in the abdomen;

• yellowness of the sclera and skin;

In the treatment of this deviation, the causes of the pathology are taken into account. The only effective treatment is surgery, since drug therapy does not give the desired result. The purpose of the operation is to ligate the portal vein. The operation is performed through the subclavian artery. It lasts 1.5–2 hours and requires the patient to stay in the hospital for about 7–10 days. In some patients, a positive result is achieved through several operations.



**Bunty Hepato-Lienal Syndrome** is a group of diseases that arise due to changes in the volume of the hepatic circle. The syndrome is named after the Italian anatomist Banta Gaspari, who first described the condition. In this article we will look at the main characteristics of banty-hepatolenal syndrome, its causes and methods of treatment.

**Definition of Bunty Hepto-Liveral Syndrome** Banty geopotential syndrome (BGS) is a pathological increase in blood volume that occurs due to impaired blood transport through the spleen and liver. This condition can lead to complications such as heart failure and can also lead to the death of the patient. GHD is one of the causes of acute cardiovascular failure (ACHF), which is classified as disseminated intravascular coagulation (DIC). This condition is classified as class three in the International Classification of Diseases (ICD-10) and class four