Gilbert-Meulengracht syndrome is a syndrome characterized by an abnormal increase in bilirubin levels in the blood. Gilbert-Meilengracht is associated with certain genes and is inherited in an autosomal recessive manner. Gilbert's disease can be mild or resemble jaundice. It may also be accompanied by other symptoms such as weakness, fatigue, headache, etc. Gilbert Meulenghrt syndrome causes abnormally elevated levels of bilirubin, a yellow pigment in the blood that is formed as a result of the breakdown of hemoglobin. Patients with Gilbert's syndrome have problems removing bilirubin from the liver into the blood, which leads to its accumulation in the body. An increase in bilirubin levels can cause a variety of symptoms and signs, such as yellowing of the skin, sclera of the eyes, mucous membranes, and jaundiced urine and stool. Symptoms may vary
Gilbert-Meulengrcht syndrome (GMS) is a hereditary benign bilirubinuria, manifested only by a paroxysmal increase in the level of indirect bilirubin in the blood, due to the pathological activity of liver and gallbladder enzymes. Etiological factors: hereditary, metabolic defect, Gilbert's triad, morphology of heredity - a sign of a cryptid.