Osteodysplasia Fibrous Polyostotic

Osteodysplasia fibrous polyostotic (OPP) is a rare hereditary disease that is characterized by damage to bone tissue, disruption of its structure and function. GPP can be either congenital or acquired.

GPP manifests itself in the form of various symptoms such as bone pain, bone deformation, limited joint mobility, and changes in the shape and size of bones. At the same time, OFP can lead to serious complications, such as skeletal deformation, dysfunction of joints, chest deformation, etc.

To diagnose AFP, various research methods are used, such as radiography, computed tomography, magnetic resonance imaging, etc. Treatment of AFP depends on the severity of the disease and may include surgery, drug therapy and orthopedic interventions.

In general, AFP is a serious disease that requires an integrated approach to treatment and prevention.

Fibrous-droplet, fibrocartilaginous or modioculoma osteodystrophy is the general name for benign tumor-like degeneration of the femur, humerus, tibia and other bones. In the vast majority of cases, they occur in the places where normal bone tissue transitions into cartilaginous tissue, that is, in the area of ​​the sinus. The tumors resemble a yellowish bubble filled with liquid contents, which seem to stretch the bone from the inside. As the tumor grows, its cavity can fill more and more of the bone, and sometimes the entire volume.

The disease progresses slowly, over years, and sometimes even decades. In adolescents, starting from puberty, degenerative processes in the bones become noticeable. The first symptom of a deformed disease is often pain, weakness in the affected limb and its unsteadiness when walking. But a doctor is consulted when a cyst forms in the area of ​​the hip or knee joint, which tends to grow into a healthy bone, after which bone deformation begins.

Fibrous osteodystrophy is a deforming disease characterized by progressive structural changes in bone tissue. This disease is characterized by irreversible damage to the structure of bone elements. Leads to severe impairment of the patient’s musculoskeletal function and serious consequences for other organs and systems. Complications arise due to delays in timely and adequate treatment. The main role in the diagnosis of osteodystrophy is given to x-ray examination, which can help determine the nature, extent and localized changes in the bones. You also cannot do without additional studies: osteoscintigraphy, MRI and CT.

The reasons for the development of osteodystrophic processes are considered to be a lack of vitamins, which provokes changes in the properties of the bone matrix. Also the consequences are problems with bone marrow tissue, developing under the influence of metabolic abnormalities and disturbances in the metabolism of calcium and phosphorus. Often such processes occur as a result of endocrine diseases, in particular hypothyroidism. Dystrophic changes in bone tissue are also caused by digestive disorders, protein deficiency, as well as chronic inflammatory bone lesions. Often, diseases of the musculoskeletal system are accompanied by convulsions, hypothermia and infectious diseases. A lack of ascorbic acid in the body, an increase in uric acid and methanol in the blood, rickets - all these disorders affect the condition of osteodystromia. Treatment is aimed at eliminating the main cause of the disease - hypocalcemia and preventing further development of bone pathology by normalizing the physicochemical properties of the bone structure. Proper nutrition and the use of pharmacological agents play an important role here.